Friday, August 21, 2020

Covid-19 Recovery?

Tenforde MW, Kim SS, Lindsell CJ, et al. Symptom Duration and Risk Factors for Delayed Return to Usual Health Among Outpatients with COVID-19 in a Multistate Health Care Systems Network — United States, March–June 2020. MMWR Morb Mortal Wkly Rep 2020;69:993-998. DOI: icon.

Return to Usual State of Health 

  • Among the 270 of 274 interviewees with available data on return to usual health,† 175 (65%) reported that they had returned to their usual state of health a median of 7 days (IQR = 5–12 days) from the date of testing (Table 1).
  • Ninety-five (35%) reported that they had not returned to their usual state of health at the time of interview.
  • The proportion who had not returned to their usual state of health differed across age groups:
    26% of interviewees aged 18–34 years, 32% aged 35–49 years,
  • 47% aged ≥50 years reported not having returned to their usual state of health (p = 0.010) within 14–21 days after receiving a positive test result.

•Presence of chronic conditions also affected return to health rates; among 180 persons with no or one chronic medical condition, 39 with two chronic medical conditions, and 44 with three or more chronic medical conditions, 28%, 46%, and 57%, respectively, reported not having returned to their usual state of health (p = 0.003) within 14–21 days after having a positive test result.

*Among respondents aged 18–34 years with no chronic medical condition, 19% (nine of 48) reported not having returned to their usual state of health




  1. The entire subfield of intellectual history is just a list of conspiracy theories.

    Also did you know that lockdowns cause coronavirus transmission?

    How can you have a 'causality problem' when everything causes everything?

    That sounds rad, I knew I went into the wrong field

    The subfield of intellectual histories is depolarizing! We're losing doxastic cohesion!! PREPARE FOR HARD LANDING INTO THE NATURAL SCIENCES!!! REIFY CRASH SAFETY THROUGH CULTURAL NORMS!!!

    If I was an intellectual I would throw off the intellectual historians by insisting I had no influences and arrived at everything organically. Really cover my tracks

    like you’ve read the “entire subfield of intellectual history.”

    1. Majia is just tracking data points to try and understand the full nature of the virus.

      If you remain unconvinced concerning the voracity of the virus perhaps we can run a little experiment for Red states that can keep their schools open this fall no matter what happens.

      Put up or shut up. Is that too intellectual for you?

    2. Ancient Humans Sweat Lodge song

      When The Song Comes, Sing it
      Listening with our heart that knows the joy
      Clean pure streams

  2. The main thing is to keep a good level of fear. Wearing a mask as much as possible even at home by yourself will remind you of the dangers. Avoid social contacts and practice being a hermit. An eremite. Read the dictionary. Find synonyms for terror.

    Listen to Joe Biden's speech over and over; and then play it backwards for the hidden meaning.

    1. LOL Yep a long continual run of fear and uncertainty, mixed messages, quite necessary for programming the next phase. lookup MKUltra. Rolled out on the entire population all at once!!

    2. By many of the same people who surround and bankroll donald trump. What a joke nukepro is

    3. Is that wat happened to the stock? One too many ketamine therapies? It just repeats same tired old politically motivated, conspiracy theories again and again.

      Follish repitition is the hobgoblin of small minds adored by racist totalitarians and violent, right wing wacko trash alike

  3. The good cops and the bad cops Stone Nuland Adelson The Bushes Pompeo Abrams
    Up is down White is Black East is west Hes the best

    1. There is the CIA and DHS NSA They pretend that there is a good side like the DNC democrats side, and a bad side like the Neoconservative Trump-nixon side . They are all in it together. It is what makes our country more of an imperial plutocracy, than a democratic republic.
      Stone is up to his ears in the deep state. Trumps closest adviser.

      Bannon is a cia, naval intelligence man.
      Trump worships and admires people, like Dulles and Angleton. They are some of his heroes. THEY MADE AMERICA GREAT according to him.
      Roy Cohn was his hero. Trump loves the NSA. He loves the DHS.
      He probably admires MK ultra for its malevolent nature. He appounted two serial torturers and pathological liars, as the cia heads. They are all serial torturers. They are all pathological liars..It is in their job descriptions. Valery plame was no saint . Nuke pro is such total joke

  4. I warn the grifters and bozoos that i carry a can of silly strung around with me.

    1. Do not forget Donny's CIA queen of torture Gina Haspel

    2. The zika hoax was a con to coverup fukushima nucleating the planet. The covid is the culmination of genetically manipulated biological warfare that started long ago.


      Fbi in action

    What is in wildfire smoke and why it is so bad for your lungs

  6. "The Questions That Everyone Asks"


    Extrapulmonary complications of COVID‐19: A multisystem disease?
    Kenneth I. Zheng MD Gong Feng MD Wen‐Yue Liu MD … See all authors
    First published: 10 July 2020
    Co‐first authors: Kenneth I. Zheng and Gong Feng.

    The outbreak of coronavirus disease 2019 (COVID‐19), caused by the severe acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2), has been recently declared a pandemic by the World Health Organization. In addition to its acute respiratory manifestations, SARS‐CoV‐2 may also adversely affect other organ systems. To date, however, there is a very limited understanding of the extent and management of COVID‐19‐related conditions outside of the pulmonary system. This narrative review provides an overview of the current literature about the extrapulmonary manifestations of COVID‐19 that may affect the urinary, cardiovascular, gastrointestinal, hematological, hematopoietic, neurological, or reproductive systems. This review also describes the current understanding of the extrapulmonary complications caused by COVID‐19 to improve the management and prognosis of patients with COVID‐19.

    SARS‐CoV‐2 appears to adversely affect not only the respiratory system but also several other organ systems, including the urinary, cardiovascular, GI, and neurological systems.

    To date, however, there is very limited understanding of the extent and management of COVID‐19‐related conditions outside of the pulmonary system.

    Further research is needed to better understand the underlying mechanisms linking SARS‐CoV‐2 with the occurrence of multiple extra‐pulmonary complications.

    The spread of coronavirus disease 2019 (COVID‐19), caused by severe acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2), has recently become a global pandemic and public health problem in almost all countries.1, 3 SARS‐CoV‐2 is similar to severe acute respiratory syndrome coronavirus (SARS) and Middle East respiratory syndrome coronavirus in that these coronavirus infections are responsible for severe and potentially life‐threatening acute respiratory syndromes in humans. As of 16 June 2020, a total of more than 7 900 000 confirmed cases and approximately 434 796 total deaths for COVID‐19 had been reported globally. Unfortunately, there are no targeted drugs for treatment of SARS‐CoV‐2 infection to date, vaccine development is at an early stage, and the number of infected patients is increasing rapidly worldwide.

  8. There is a growing body of evidence suggesting that in addition to the common acute respiratory symptoms (such as fever, cough, and dyspnea), COVID‐19 patients may also have signs and symptoms of injury in many other organ systems (as summarized in Figure 1), which may further complicate medical management and adversely affect clinical outcomes of these patients.

    SARS‐CoV‐2 is thought to use cell receptor angiotensin‐converting enzyme 2 (ACE2) to gain cellular access in humans.5 The ACE2 receptor is highly expressed in lungs, kidneys, gastrointestinal (GI) tract, liver, vascular endothelial cells, and arterial smooth muscle cells.6 Thus, all of these organs and systems with high expression of ACE2 receptors might be speculated targets for SARS‐CoV‐2 infection.7

    The main purpose of this narrative review article is to provide an overview of the current literature on the extrapulmonary manifestations and complications of COVID‐19 to improve the management and prognosis of these patients.

    The diagnosis of SARS‐CoV‐2 infection is currently established with nucleic acid (RNA) testing of suspected patients using real‐time reverse transcriptase‐polymerase chain reaction (RT‐PCR) techniques by oropharyngeal swabs or, in some cases, by stool samples.8, 9 Initially, a patient was suspected of SARS‐CoV‐2 infection if he/she had symptoms of cough, fever and/or dyspnea, and a history of travel to endemic regions affected by the SARS‐CoV‐2 outbreak; or have had close contact(s) with individuals with an aforementioned travel history. However, due to the ever‐increasing number of COVID‐19 cases, physicians are now recommending RT‐PCR testing only in all patients showing any evidence of viral pneumonia on chest X‐ray or computed tomography (CT) (eg, ground‐glass opacities and exudative lesions).10, 12 In some cases, absence of fever and typical symptoms in the early stages of viral infection hinders the identification of infection in at‐risk individuals.13 To date, the treatment options are scarce, mostly due to the fact that no targeted therapy for SARS‐CoV‐2 is available. The mainstay of COVID‐19 management is the patient's isolation and supportive medical care, as recommended by National Institutes of Health of the United States and China Center for Disease Control and

    1. Prevention, which includes the use of antiviral, antibacterial medications, and oxygenation therapy as appropriate.14, 15 Initially, corticosteroids were not recommended for routine use as their usage may be associated with delayed viral clearance.16 However, the latest trial data demonstrated that low to moderate dosage of dexamethasone may reduce mortality by 20% among critically ill patients, especially for patients requiring ventilation therapy.17 (For the latest updated treatment and management of severe COVID‐19 see15). The incidence of acute respiratory distress syndrome (ARDS) in COVID‐19 patients is reported to be 15% to 30%.18, 20 Compared to survivors, patients dying with COVID‐19 are more likely to be older, have more severe viral infection, be admitted to intensive care unit (ICU), and are more likely to have comorbidities or develop ARDS. For survivors, the median recovery time from hospital admission to discharge is approximately 12 to 14 days21, 22; the median duration from ICU admission to death for nonsurvivors is approximately 7 days.23 The discharge criteria for COVID‐19 patients after in‐hospital treatment varied across the globe, while specific criteria can be quickly evolving (for detailed comparison between guidelines, please see).24

      The kidneys are one of the most frequently affected extrapulmonary organs in patients infected with SARS‐CoV‐2; especially, in those patients who are severely ill.18, 20, 23 Previous studies of patients affected by the 2013 SARS outbreak have shown that kidney damage is mainly characterized by tubular injury (as reflected by abnormal urine test results) and increased serum creatinine and urea nitrogen concentrations.25, 26 A recent study of 59 patients infected with SARS‐CoV‐2 (nearly half of whom had a severe illness) showed that mild proteinuria was the commonest kidney abnormality in these patients. In addition, nearly 30% of these patients also had elevated urea nitrogen levels and approximately 20% had increased serum creatinine levels.27

  9. 1000 more words to abstract

  10. I did not know anyone could spit out this many lies, so fast. Frontline ppe ventilators ? It is amazing how jazzed up people, are on a few lines of adderall. It doesn't do anything for his sub-normal iq



Note: Only a member of this blog may post a comment.